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It binds into the spike - the corona - protein, making it unable to key into ACE-2 receptors at the surface of the cells and making it open https://pubmed.ncbi.nlm.nih.gov/32871846/ It’s like a key smeared with epoxy that has hardened, it cannot fit into the keyhole anymore. This includes emerging strains with mutations on the spike protein that threaten to disable existing vaccines. Unlike the vaccines, this allows the immune system to develop antibodies – immunity - for each emerging variant spike protein encountered https://www.who.int/news-room/feature-stories/detail/how-do-vaccines-work

It binds into the spike - the corona - protein, making it unable to key into ACE-2 receptors at the surface of the cells and making it open https://pubmed.ncbi.nlm.nih.gov/32871846/ It’s like a key smeared with epoxy that has hardened, it cannot fit into the keyhole anymore. This includes emerging strains with mutations on the spike protein that threaten to disable existing vaccines. Unlike the vaccines, this allows the immune system to develop antibodies – immunity - for each emerging variant spike protein encountered https://www.who.int/news-room/feature-stories/detail/how-do-vaccines-work

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Ivermectin has many mechanisms of actions, and blocking the virus is not the main MOA. The virus sends proteins into the nucleus to suppress the gene expression needed for self defence. Ivermectin blocks the importer molecules used by the virus, allowing infected cells to inform the immune system about the menace. Another mechanism helps fight the cytokine storm, making ivermectin useful also in late stages of C19.